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Expression of sodium channel α subunits 1.1, 1.2 and 1.6 in rat hippocampus after kainic acid-induced epilepsy
Epilepsy research, 2013-09, Vol.106 (1), p.17-28
2013

Details

Autor(en) / Beteiligte
Titel
Expression of sodium channel α subunits 1.1, 1.2 and 1.6 in rat hippocampus after kainic acid-induced epilepsy
Ist Teil von
  • Epilepsy research, 2013-09, Vol.106 (1), p.17-28
Ort / Verlag
Kidlington: Elsevier B.V
Erscheinungsjahr
2013
Link zum Volltext
Quelle
Elsevier ScienceDirect Journals Complete
Beschreibungen/Notizen
  • Highlights • NaV 1.1, NaV 1.2 and NaV 1.6 expression were investigated during epileptogenesis. • NaV 1.1 and NaV 1.6 were persistently reduced during epileptogenesis. • NaV 1.2 in the DG molecular layer disappeared acutely, but gradually recovered. • The recovery of NaV 1.2 in the molecular layer correlated with mossy fiber sprouting. • Distinct changes of NaV occur during epileptogenesis which will affect excitability.
Sprache
Englisch
Identifikatoren
ISSN: 0920-1211
eISSN: 1872-6844
DOI: 10.1016/j.eplepsyres.2013.06.006
Titel-ID: cdi_proquest_miscellaneous_1429639676
Format
–
Schlagworte
Animals, Biological and medical sciences, CA1 Region, Hippocampal - metabolism, CA3 Region, Hippocampal - metabolism, Convulsants, Data Interpretation, Statistical, Electrodes, Implanted, Electroencephalography - drug effects, Epilepsy - chemically induced, Epilepsy - metabolism, Epilepsy - pathology, Epileptogenesis, Excitatory Amino Acid Agonists, Fluorescent Antibody Technique, Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy, Hippocampus - drug effects, Hippocampus - metabolism, Hippocampus - pathology, Immunohistochemistry, Interneurons - metabolism, Kainic Acid, Male, Medical sciences, Mossy fiber sprouting, NAV1.1 Voltage-Gated Sodium Channel - biosynthesis, NAV1.1 Voltage-Gated Sodium Channel - drug effects, NAV1.2 Voltage-Gated Sodium Channel - biosynthesis, NAV1.2 Voltage-Gated Sodium Channel - drug effects, NAV1.6 Voltage-Gated Sodium Channel - biosynthesis, NAV1.6 Voltage-Gated Sodium Channel - drug effects, Nerve Tissue Proteins - metabolism, Nervous system (semeiology, syndromes), Neurology, Rats, Rats, Sprague-Dawley, Seizures - physiopathology, Status epilepticus, Status Epilepticus - chemically induced, Status Epilepticus - physiopathology, Temporal lobe epilepsy

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