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Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

Circulation research, 2010-04, Vol.106 (7), p.1253-1264

2010

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Mitochondrial NADH Kinase, Pos5p, Is Required for Efficient Iron-Sulfur Cluster Biogenesis in Saccharomyces cerevisiae

The Journal of biological chemistry, 2010-12, Vol.285 (50), p.39409-39424

2010

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Aging increases stiffness of cardiac myocytes measured by atomic force microscopy nanoindentation

American journal of physiology. Heart and circulatory physiology, 2004-08, Vol.287 (2), p.H645-H651

2004

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Adenylyl cyclase type 5 protein expression during cardiac development and stress

American journal of physiology. Heart and circulatory physiology, 2009-11, Vol.297 (5), p.H1776-H1782

2009

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Mitochondria export iron–sulfur and sulfur intermediates to the cytoplasm for iron–sulfur cluster assembly and tRNA thiolation in yeast

The Journal of biological chemistry, 2019-06, Vol.294 (24), p.9489-9502

2019

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Nfs1 cysteine desulfurase protein complexes and phosphorylation sites as assessed by mass spectrometry

Data in brief, 2017-12, Vol.15, p.775-799

2017

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Essential mitochondrial role in iron-sulfur cluster assembly of the cytoplasmic isopropylmalate isomerase Leu1 in Saccharomyces cerevisiae

Mitochondrion, 2023-03, Vol.69, p.104-115

2023

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Frataxin Directly Stimulates Mitochondrial Cysteine Desulfurase by Exposing Substrate-binding Sites, and a Mutant Fe-S Cluster Scaffold Protein with Frataxin-bypassing Ability Acts Similarly

The Journal of biological chemistry, 2013-12, Vol.288 (52), p.36773-36786

2013

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Characterization of a Novel Cardiac Isoform of the Cell Cycle-related Kinase That Is Regulated during Heart Failure

The Journal of biological chemistry, 2008-08, Vol.283 (32), p.22157-22165

2008

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Mitochondria Export Sulfur Species Required for Cytosolic tRNA Thiolation

Cell chemical biology, 2018-06, Vol.25 (6), p.738-748.e3

2018

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Fe-S cluster biogenesis in isolated mammalian mitochondria: coordinated use of persulfide sulfur and iron and requirements for GTP, NADH, and ATP

The Journal of biological chemistry, 2015-01, Vol.290 (1), p.640-657

2015

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Frataxin-bypassing Isu1: characterization of the bypass activity in cells and mitochondria

Biochemical journal, 2014-04, Vol.459 (1), p.71-81

2014

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Splitting the functions of Rim2, a mitochondrial iron/pyrimidine carrier

Mitochondrion, 2019-07, Vol.47, p.256-265

2019

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Rim2, a pyrimidine nucleotide exchanger, is needed for iron utilization in mitochondria

Biochemical journal, 2011-11, Vol.440 (1), p.137-146

2011

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Frataxin or a mutant Fe‐S cluster scaffold protein with frataxin‐bypassing ability directly stimulates mitochondrial cysteine desulfurase by exposing substrate‐binding sites (578.4)

The FASEB journal, 2014-04, Vol.28 (S1), p.n/a

2014

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Cysteine desulfurase is regulated by phosphorylation of Nfs1 in yeast mitochondria

Mitochondrion, 2018-05, Vol.40, p.29-41

2018

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Endogenous Muscle Atrophy F-Box Mediates Pressure Overload–Induced Cardiac Hypertrophy Through Regulation of Nuclear Factor-κB

Circulation research, 2011-07, Vol.109 (2), p.161-171

2011

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Fe-S Cluster Biogenesis in Isolated Mammalian Mitochondria

The Journal of biological chemistry, 2015-01, Vol.290 (1), p.640-657

2015

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Mutation in the Fe-S scaffold protein Isu bypasses frataxin deletion

Biochemical journal, 2012-01, Vol.441 (1), p.473-480

2012

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Turning Saccharomyces cerevisiae into a Frataxin-Independent Organism: e1005135

PLoS genetics, 2015-05, Vol.11 (5)

2015

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In vitro characterization of a novel Isu homologue from Drosophila melanogaster for de novo FeS-cluster formation

Metallomics, 2017-01, Vol.9 (1), p.48

2017

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Histone H2A.z Is Essential for Cardiac Myocyte Hypertrophy but Opposed by Silent Information Regulator 2α

The Journal of biological chemistry, 2006-07, Vol.281 (28), p.19369-19377

2006

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Mutation in Fe-S scaffold Isu bypasses frataxin deletion

Biochemical journal, 2011-09

2011

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Abstract 1367: The Nad(p)h Oxidase Component Nox4 Is Partially Localized In Mitochondria In Cardiac Myocytes

Circulation (New York, N.Y.), 2008-10, Vol.118 (suppl_18)

2008

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Endogenous Muscle Atrophy F-box Mediates Pressure Overload-Induced Cardiac Hypertrophy through Regulation of NF-κB

Circulation research, 2011-05, Vol.109 (2), p.161-171

2011

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Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

Mitochondrial NADH Kinase, Pos5p, Is Required for Efficient Iron-Sulfur Cluster Biogenesis in Saccharomyces cerevisiae

Aging increases stiffness of cardiac myocytes measured by atomic force microscopy nanoindentation

Adenylyl cyclase type 5 protein expression during cardiac development and stress

Mitochondria export iron–sulfur and sulfur intermediates to the cytoplasm for iron–sulfur cluster assembly and tRNA thiolation in yeast

Nfs1 cysteine desulfurase protein complexes and phosphorylation sites as assessed by mass spectrometry

Essential mitochondrial role in iron-sulfur cluster assembly of the cytoplasmic isopropylmalate isomerase Leu1 in Saccharomyces cerevisiae

Frataxin Directly Stimulates Mitochondrial Cysteine Desulfurase by Exposing Substrate-binding Sites, and a Mutant Fe-S Cluster Scaffold Protein with Frataxin-bypassing Ability Acts Similarly

Characterization of a Novel Cardiac Isoform of the Cell Cycle-related Kinase That Is Regulated during Heart Failure

Mitochondria Export Sulfur Species Required for Cytosolic tRNA Thiolation

Fe-S cluster biogenesis in isolated mammalian mitochondria: coordinated use of persulfide sulfur and iron and requirements for GTP, NADH, and ATP

Frataxin-bypassing Isu1: characterization of the bypass activity in cells and mitochondria

Splitting the functions of Rim2, a mitochondrial iron/pyrimidine carrier

Rim2, a pyrimidine nucleotide exchanger, is needed for iron utilization in mitochondria

Frataxin or a mutant Fe‐S cluster scaffold protein with frataxin‐bypassing ability directly stimulates mitochondrial cysteine desulfurase by exposing substrate‐binding sites (578.4)

Cysteine desulfurase is regulated by phosphorylation of Nfs1 in yeast mitochondria

Endogenous Muscle Atrophy F-Box Mediates Pressure Overload–Induced Cardiac Hypertrophy Through Regulation of Nuclear Factor-κB

Fe-S Cluster Biogenesis in Isolated Mammalian Mitochondria

Mutation in the Fe-S scaffold protein Isu bypasses frataxin deletion

Turning Saccharomyces cerevisiae into a Frataxin-Independent Organism: e1005135

In vitro characterization of a novel Isu homologue from Drosophila melanogaster for de novo FeS-cluster formation

Histone H2A.z Is Essential for Cardiac Myocyte Hypertrophy but Opposed by Silent Information Regulator 2α

Mutation in Fe-S scaffold Isu bypasses frataxin deletion

Abstract 1367: The Nad(p)h Oxidase Component Nox4 Is Partially Localized In Mitochondria In Cardiac Myocytes

Endogenous Muscle Atrophy F-box Mediates Pressure Overload-Induced Cardiac Hypertrophy through Regulation of NF-κB

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