Details

Autor(en) / Beteiligte

• Bramer, Lisa M
• Dixon, Holly M
• Rohlman, Diana
• Scott, Richard P
• Miller, Rachel L
• Kincl, Laurel
• Herbstman, Julie B
• Waters, Katrina M
• Anderson, Kim A

Titel

PM 2.5 Is Insufficient to Explain Personal PAH Exposure

Ist Teil von

• Geohealth, 2024-02, Vol.8 (2), p.e2023GH000937

Ort / Verlag

United States

Erscheinungsjahr

2024

Quelle

Wiley Online Library Journals Frontfile Complete

Beschreibungen/Notizen

• To understand how chemical exposure can impact health, researchers need tools that capture the complexities of personal chemical exposure. In practice, fine particulate matter (PM ) air quality index (AQI) data from outdoor stationary monitors and Hazard Mapping System (HMS) smoke density data from satellites are often used as proxies for personal chemical exposure, but do not capture total chemical exposure. Silicone wristbands can quantify more individualized exposure data than stationary air monitors or smoke satellites. However, it is not understood how these proxy measurements compare to chemical data measured from wristbands. In this study, participants wore daily wristbands, carried a phone that recorded locations, and answered daily questionnaires for a 7-day period in multiple seasons. We gathered publicly available daily PM AQI data and HMS data. We analyzed wristbands for 94 organic chemicals, including 53 polycyclic aromatic hydrocarbons. Wristband chemical detections and concentrations, behavioral variables (e.g., time spent indoors), and environmental conditions (e.g., PM AQI) significantly differed between seasons. Machine learning models were fit to predict personal chemical exposure using PM AQI only, HMS only, and a multivariate feature set including PM AQI, HMS, and other environmental and behavioral information. On average, the multivariate models increased predictive accuracy by approximately 70% compared to either the AQI model or the HMS model for all chemicals modeled. This study provides evidence that PM AQI data alone or HMS data alone is insufficient to explain personal chemical exposures. Our results identify additional key predictors of personal chemical exposure.